(HOME) Subject: stroke/iron
Iron levels linked to stroke damage
April 25, 2000
Reuters
NEW YORK, Apr 24 (Reuters Health) - People with high levels of
iron in their bodies may be at higher risk for severe damage to the
brain after a stroke, according to study results released Monday.
People at high risk of having a stroke who have high iron
levels might benefit from cutting back on iron in the diet, according
to the study's lead author, Dr. Antoni Davalos.
An ischemic stroke -- the most common type of stroke -- is
caused by a disruption in blood flow to the brain, often due to a
blocked artery in the neck or head.
Some research has linked iron levels to the severity of stroke
damage, so Davalos, of the Hospital Universitari Doctor Josep Trueta
in Girona, Spain, and colleagues measured levels of ferritin (which
indicates the total amount of iron in the body) in the spinal fluid
and blood of 100 stroke patients. The investigators divided the
patients into two groups -- those whose condition remained stable or
began to improve after the stroke, and those whose condition
progressively worsened.
Compared with people whose condition stabilized or improved,
those with progressive damage had significantly higher levels of
ferritin, the authors report in the April 25th issue of Neurology, the
journal of the American Academy of Neurology.
Davalos and colleagues suspect that increased iron levels lead
to greater brain damage by increasing the formation of molecules
called free radicals, which can damage cells. In addition, high levels
of iron can promote the release of glutamate, a brain chemical
involved in the death of brain cells. In the study, glutamate levels
were highest in people with progressing stroke.
"Our findings support future therapeutic studies of agents
which inhibit iron's toxic effects on brain cells immediately after
stroke," Davalos said in a statement. Studies should also be conducted
to see whether lowering iron levels in people at risk for stroke who
have high ferritin levels might help prevent stroke, he noted.
SOURCE: Neurology 2000;54:1568-1574.
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